Hauptinhalt

Zehn ausgewählte Publikationen

• Decher N, Ortiz-Bonnin B, Friedrich C, Schewe M, Kiper AK, Rinné S, Seemann G, Peyronnet R, Zumhagen S, Bustos D, Kockskämper J, Kohl P, Just S, González W, Baukrowitz T, Stallmeyer B. & Schulze-Bahr E. (2017).
  Sodium permeable and `hypersensitive´ TREK-1 channels cause ventricular tachycardia.
  EMBO Molecular Medicine, 9: 403-414. doi: 10.15252/emmm.201606690
  
  
• Stallmeyer B, Kuß J, Kotthoff S, Zumhagen S, Vowinkel K, Rinné S, Matschke LA, Friedrich C, Schulze-Bahr E, Rust S, Seebohm G, Decher N & Schulze-Bahr E. (2017).
  A mutation in the G-protein gene GNB2 causes familial sinus node and atrioventricular conduction dysfunction.
  Circulation Research, 120: e33-e44. doi: 10.1161/CIRCRESAHA.116.310112
  
  
• Schindler RFR, Scotton C, Zhang J, Passarelli C, Ortiz-Bonnin B, Simrick S, Schwerte T, Poon K-L, Fang M, Rinné S, Froese A, Nikolaev VO, Grunert C, Müller T, Tasca G, Sarathchandra P, Drago F, Dallapiccola B, Rapezzi C, Arbustini E, Di Raimo FR, Neri M, Selvatici R, Gualandi F, Fattori F, Pietrangelo A, Li W, Jiang H, Xu X, Bertini E, Decher N, Wang J, Brand T & Ferlini A. (2016).
  POPDC1S201F causes muscular dystrophy and arrhythmia by affecting protein trafficking.
  The Journal of Clinical Investigation, 126: 239-53. doi: 10.1172/JCI79562
  
  
• Friedrich C, Rinné S, Zumhagen S, Kiper AK, Silbernagel N, Netter MF, Stallmeyer B, Schulze-Bahr E & Decher N. (2014).
  Gain-of-function mutation in TASK-4 channels and severe cardiac conduction disorder.
  EMBO Molecular Medicine, 6: 937-951. doi: 10.15252/emmm.201303783
  
  
• Marzian S, Stansfeld PJ, Rapedius M, Rinné S, Nematian-Ardestani E, Abbruzzese JL, Steinmeyer K, Sansom MSP, Sanguinetti MC, Baukrowitz T & Decher N. (2013).
  Side pockets provide the basis for a new mechanism of Kv channel–specific inhibition.
  Nature Chemical Biology, 9: 507-513. doi: 10.1038/nchembio.1271
  
  
• Froese A, Breher SS, Waldeyer C, Schindler RF, Nikolaev VO, Rinné S, Wischmeyer E, Schlueter J, Becher J, Simrick S, Vauti F, Kuhtz J, Meister P, Kreissl S, Torlopp A, Liebig SK, Laakmann S, Müller TD, Neumann J, Stieber J, Ludwig A, Maier SK,Decher N, Arnold HH, Kirchhof P, Fabritz L & Brand T (2012).
  Popeye domain containing proteins are essential for stress-mediated modulation of cardiac pacemaking in mice.
  The Journal of Clinical Investigation, 122: 1119-30. doi: 10.1172/JCI59410
  
  
• Decher N, Streit AK, Rapedius M, Netter MF, Marzian S, Ehling P, Schlichthörl G, Craan T, Renigunta V, Köhler A, Dodel RC, Navarro-Polanco RA, Preisig-Müller R, Klebe G, Budde T, Baukrowitz T & Daut J. (2010).
  RNA editing modulates the binding of drugs and highly unsaturated fatty acids to the open pore of Kv potassium channels.
  The EMBO Journal, 29: 2101-2113. doi: 10.1038/emboj.2010.88
  
  
• Decher N, Gonzalez T, Streit AK, Sachse F, Renigunta V, Soom M, Heineman SH, Daut J & Sanguinetti MC. (2008).
  Structural determinants of Kvß1 induced channel inactivation: a PIP2 modulated hairpin.
  The EMBO Journal, 27: 3164-3174. doi: 10.1038/emboj.2008.231
  
  
• Splawski I, Timothy KW, Decher N, Kumar P, Sachse FB, Beggs AH, Sanguinetti MC & Keating MT. (2005).
  Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations.
  Proceedings of the National Academy of Sciences of the United States of America, 102: 8089-8096. doi: 10.1073/pnas.0502506102
  
  
• Splawski I, Timothy KW, Sharpe LM, Decher N, Kumar P, Bloise R, Napolitano C, Schwartz PJ, Joseph RM, Condouris K, Tager-Flusberg H, Priori SG, Sanguinetti MC & Keating MT. (2004).
  CaV1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism.
  Cell, 119: 19-31. doi: 10.1016/j.cell.2004.09.011